| Bildiri Türü | Tebliğ/Bildiri | Bildiri Dili | İngilizce |
| Bildiri Alt Türü | Özet Metin Olarak Yayınlanan Tebliğ (Uluslararası Kongre/Sempozyum) | ||
| Bildiri Niteliği | Web of Science Kapsamındaki Kongre/Sempozyum | ||
| DOI Numarası | 10.1016/S0735-1097(11)60118-6 | ||
| Kongre Adı | |||
| Kongre Tarihi | / | ||
| Basıldığı Ülke | Basıldığı Şehir | ||
| Bildiri Linki | https://www.jacc.org/doi/abs/10.1016/S0735-1097(11)60118-6 | ||
| UAK Araştırma Alanları |
Sağlık Bilimleri
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| Özet |
| Background: In the absence of the coronary artery disease (CAD), left ventricular hypertrophy (LVH) appears to be the main link between hypertension and ventricular arrhythmias (VA). Myocardial fibrosis is a key pathological feature of myocardial remodeling in LVH. Fragmented QRS (fQRS) on a 12-lead ECG has been shown to represent myocardial scar in patients with CAD. Myocardial fibrosis may also alter QRS morphology and lead to fragmentation of QRS on 12-lead ECG in patient with hypertension. We therefore postulated that the prevalence of fQRS is increased and it is associated with VA in hypertensive patients.Methods: Patients with newly diagnosed or previous history of hypertension were included. Age and sex matched normotensive individuals formed control group. Fragmented QRS was defined as QRS with> 1 notch in the R or the S wave on a routine 12-lead ECG. 24-hour Holter monitoring was … |
| Anahtar Kelimeler |
| Atıf Sayıları | |
| Web of Science | 1 |
| Google Scholar | 3 |
| Dergi Adı | JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY |
| Yayıncı | Elsevier Inc. |
| Açık Erişim | Evet |
| ISSN | 0735-1097 |
| E-ISSN | 1558-3597 |
| CiteScore | 42,7 |
| SJR | 8,762 |
| SNIP | 6,318 |