| Makale Türü |
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| Dergi Adı | Journal of Clinical Sleep Medicine (Q1) | ||
| Dergi ISSN | 1550-9389 Wos Dergi Scopus Dergi | ||
| Dergi Tarandığı Indeksler | SCI-Expanded | ||
| Makale Dili | İngilizce | Basım Tarihi | 08-2020 |
| Kabul Tarihi | 02-04-2026 | Yayınlanma Tarihi | 15-08-2020 |
| Cilt / Sayı / Sayfa | 16 / 8 / 1403–1404 | DOI | 10.5664/jcsm.8576 |
| Makale Linki | https://jcsm.aasm.org/doi/pdf/10.5664/jcsm.8576 | ||
| Özet |
| Because hypertension (HT) is a primary risk factor for severe disease and mortality in the ongoing coronavirus disease 2019 (COVID-19) pandemic, the renin-angiotensin system (RAS) is of much greater concern as the main underlying physiopathology of HT. 1 However, obstructive sleep apnea (OSA), repetitive episodes of complete/partial cessation of airflow (breathing) during sleep, is associated with dysregulation of the RAS, and HT is a significant comorbidity among patients with OSA. 2 Accordingly, in this report, we draw attention to the possible interactions among OSA, RAS, and COVID-19.Describing the RAS is helpful to better explain these interactions. The RAS has 2 primary pathways: classical and nonclassical. In the former, angiotensinogen is converted to angiotensin I by renin that is secreted from the kidneys. Then angiotensin I is converted to angiotensin II by the angiotensinconverting enzyme … |
| Anahtar Kelimeler |
| Atıf Sayıları | |
| Google Scholar | 34 |
| Scopus | 4 |
| Web of Science | 14 |
| Dergi Adı | Journal of Clinical Sleep Medicine |
| Yayıncı | American Academy of Sleep Medicine |
| Açık Erişim | Hayır |
| ISSN | 1550-9389 |
| E-ISSN | 1550-9397 |
| CiteScore | 6,2 |
| SJR | 1,039 |
| SNIP | 1,199 |