Evaluation of the Association between Paranasal Sinus Osteomas and Anatomic Variations Using Computed Tomography      
Yazarlar (3)
Ceyhun Aksakal
Tokat Gaziosmanpaşa Üniversitesi, Türkiye
Doç. Dr. Murat BEYHAN Tokat Gaziosmanpaşa Üniversitesi, Türkiye
Prof. Dr. Erkan GÖKÇE Tokat Gaziosmanpaşa Üniversitesi, Türkiye
Makale Türü Özgün Makale
Makale Alt Türü ESCI dergilerinde yayınlanan tam makale
Dergi Adı Turkish archives of otorhinolaryngology
Dergi ISSN 2667-7466 Wos Dergi
Dergi Tarandığı Indeksler ESCI
Makale Dili İngilizce
Basım Tarihi 01-2021
Cilt No 59
Sayı 1
Sayfalar 54 / 64
DOI Numarası 10.4274/tao.2020.5811
Makale Linki http://dx.doi.org/10.4274/tao.2021.2020.5811
Özet
Objective: The pathogenesis of paranasal sinus osteoma (PSO) has not been fully elucidated. It is
thought that both embryological and developmental factors play a role in the etiology. The aim of
the present study was to investigate the association of frequency and localization of PSOs detected
on computed tomography (CT) examination with osteoma presence.
Methods: In this retrospective study conducted in December 2017 through March 2020 in
Gaziosmanpaşa University Faculty of Medicine, images of a total of 18,867 patients who
underwent paranasal sinus, maxillofacial CT and brain CT angiography were reviewed for
the presence of PSOs. Sizes of PSOs and accompanying mucosal pathologies were identified.
Associations between PSOs and paranasal sinus variations were evaluated statistically compared
to the control group (200 patients without PSO).
Results: A total of 176 patients (0.92%) were found to have PSO. Average age of the patients with
PSO was 59.9 years (range: 18–93). PSOs were unilateral in 152 patients while 24 patients had
multiple osteomas. Female/male ratio was 1.1/1. PSOs were most commonly located in the frontal
sinuses. Frequencies of vertical concha bullosa, secondary middle turbinate, twisted uncinate,
supraorbital ethmoid cell, intersinus septal cell, ethmoidomaxillary cell, Haller’s cell, frontal sinus
hypoplasia and sphenoid sinus hypoplasia were significantly higher in the patient group compared
to the control group.
Conclusion: Higher or lower incidence rates of some anatomic variations in the patients with
PSO could be explained by the possible effects of genetic and/or environmental factor. Additional
studies are needed to evaluate these possible associations.
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